About 45 minutes ago, my 10kg (22 lb) re

I'm opening the skill file first to make sure my advice matches the protocol.

Go to the nearest emergency vet now. Don't wait.

Your dog is showing a red-flag constellation: severe ataxia, dilated/glassy eyes, urinary incontinence, hyperesthesia, and hypothermia with bradycardia/shallow breathing. Cannabis toxicity in dogs is not "sleeping it off," and the friend's peroxide plan and the roommate's wait-it-out plan are both wrong. Both put him at higher risk.

Right now, while you get him to the car:

• Call ahead to the nearest 24-hour emergency vet or your regular vet's after-hours line — they will direct you to the closest open one. Search "emergency veterinarian near me" if you don't have one already. (Tell me your city and I'll pull the closest open clinics from a map search.)
• Warm him gently — a towel or blanket warmed in the dryer, draped over the body. No direct heating pads or hot water bottles: he cannot tell you if he's burning. His thermoregulation is impaired.
• Keep him dark, quiet, and still. THC hyperesthesia makes light, sound, and touch feel excruciating. Lower car windows a crack for fresh air; no music.
• Keep his head slightly elevated and to one side so saliva and any vomit don't pool in his airway — ataxic, sedated dogs aspirate easily.
• Bring the chewed foil packet (or a photo of the package) so the vet knows the exact formulation and dose.

Do NOT force-feed hydrogen peroxide or try to make him vomit. More on why below.

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1. Why "sleep it off" is wrong

Cannabis toxicity in dogs is mechanistically different from humans, and the dose he got was significant. You described about 100 mg total THC for a 10 kg dog — roughly 10 mg/kg. Clinical signs reliably appear around 1–2 mg/kg in dogs; severe neurological signs like you're describing typically begin in the 5–10 mg/kg range.

Why it's not "just a high":

• CB1 receptor density is higher in the dog cerebellum and brainstem than in humans. Those areas control balance, postural tone, and vital autonomic functions (heart rate, breathing). That's why you see severe truncal ataxia and bradycardia, not just sedation.
• Hypothermia is a primary clinical effect, not a side effect. THC disrupts hypothalamic thermoregulation. Your observation that his ears and paws are cold, with slow/shallow breathing, is the early phase of clinically significant hypothermia — drops below 94 °F (34.5 °C) are reported and worsen bradycardia and can progress to coma.
• Bradycardia and respiratory depression can deepen over the next several hours as more THC is absorbed from the gut. "Sleeping it off" can quietly become a breathing emergency.
• Secondary risks you cannot see at home: aspiration pneumonia (he's sedated and dribbling — if he vomits while unconscious, gastric contents go into the lungs), urinary retention with bladder atony, prolonged coma, and — though rare — seizures at high doses or in dogs with prior epilepsy.
• THC also has anti-emetic properties in dogs, which is one reason peroxide-induced vomiting is dangerous: he may not bring it up, the peroxide sits in the stomach causing gastric ulceration, and if he does vomit while ataxic he aspirates.

The corpus on cannabis in pets classifies exposure as moderate-to-high toxicity requiring immediate veterinary triage, with the dose-effect relationship variable. The combination of a confirmed large dose plus the neurological signs you listed is not a watch-and-wait situation.

2. Why peroxide is dangerous, and what to do on the way

Hydrogen peroxide (3%) is contraindicated here, despite the friend's advice:

• Aspiration risk. He is heavily sedated and ataxic. Emesis in a dog with depressed airway reflexes causes gastric contents to enter the lungs — aspiration pneumonia can be more lethal than the THC.
• THC is anti-emetic in dogs. Peroxide may not produce vomiting at all; the peroxide itself then irritates and can ulcerate the gastric mucosa.
• Time window already passed. Most of the dose has been absorbed in 45 minutes; induced vomiting at this stage retrieves little and adds risk.
• Other ingredients may matter. Edible gummies sometimes contain xylitol (a separate, fast-acting hypoglycemia and liver-failure toxin in dogs) or chocolate. The vet needs the package to check this.

On the drive:

• Drive calm and quiet. Lights low. A crate or box lined with a blanket keeps him stable; if he's too ataxic to be in a crate, hold his body stable with someone next to him in the back seat.
• Do not offer food, water, or milk.
• If he vomits, turn his head down and to the side and clear his mouth.
• If breathing becomes very slow or stops, that is a true respiratory arrest — call the vet clinic on the phone for instructions while you drive; do not stop driving to attempt CPR unless you are on the phone with the vet.

3. What the ER vet will actually do, and the legal reality

Typical ER protocol for confirmed/recent cannabis ingestion with neurological signs (this is the standard of care, not invented for this reply):

• IV fluids to maintain perfusion and help with thermoregulation, especially if his rectal temp is low.
• Active warming — warmed IV fluids, blankets, controlled warming devices — to bring core temperature back up safely.
• Continuous monitoring of heart rate/rhythm, respiratory rate, blood pressure, temperature, and mentation.
• Activated charcoal with a cathartic, if it's safe to administer and the airway is protected (often given once, sometimes repeated). For a 10 kg dog who got ~100 mg, this is reasonable decontamination.
• Intralipid (IV lipid emulsion) therapy — increasingly used for severe lipophilic-toxin overdoses including cannabis; this is a decision the ER vet makes based on severity.
• Oxygen supplementation if breathing becomes inadequate.
• Hospitalization for observation — clinical signs can persist 18–72 hours; most dogs survive with supportive care, but they need to be watched through the peak.

Cost transparency (since your roommate raised this): emergency exam + IV fluids + monitoring + charcoal typically runs $400–$900 depending on your city. Intralipid, hospitalization overnight, and oxygen can push it higher. This is a legitimate medical intervention, not an upcharge. For a 10 kg dog with the signs you're describing, this is the appropriate level of care.

Legal / disclosure reality:

• In most U.S. states, veterinarians do NOT report THC ingestion to law enforcement. Veterinary practice acts in most jurisdictions include a confidentiality duty, and cannabis exposure is treated as a medical-toxicology case, not a reporting trigger (unlike, for example, suspected dog fighting or repeated malicious animal harm, which some states do require reporting).
• Tell the vet exactly what he ate, how many, and when. "Sleeping it off" by the owner's description delays diagnosis and can send the vet down a much more expensive and invasive path — MRI to rule out stroke, full bloodwork to rule out metabolic causes, toxicology panels. Plain disclosure that he ate 10 × 10 mg THC gummies 45 minutes ago lets the vet focus on the cannabis protocol and skip the stroke rule-out.
• The vet is on your side medically, not legally. The staff's job is to treat the dog.

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The single most important thing right now is to drive to the nearest emergency vet. If you tell me your city or district, I can pull the closest open clinics with phone numbers from a map search while you get him in the car.